Question for OP:
since Jesus rode dinosaurs, which do you think was his favorite to keep as a pet?
I have no idea since Jesus is likely a figment of man's imagination. ()
Question for OP:
since Jesus rode dinosaurs, which do you think was his favorite to keep as a pet?
Any way, the reason why your explanation doesn't make sense to me, is that it doesn't account for the fact that for mutation to be responsible for such an elaborate and well ordered system (if you read the article, synethesis of the flagellum is dependent on a "tightly ordered cascade in which expression of one gene at a given level requires the prior expression of another gene at a higher level") as bacterial flagella, all the right mutations (and none of the harmful ones) would have to occur at the same time without mistakes, instead of the numerous, slight successive changes ascribed to by darwinian evolution.
I have no idea since Jesus is likely a figment of man's imagination. ()![]()
The rate doesn't have to be even I agree, but there should be far more transitional stages in the fossil record before the cambrian explosion.
We're talking billions of years here, yet the fossil record doesn't indicate any sort of major or minor transitional changes that occurred during the time before the cambrian era.
There's a reason for that: they haven't been trying. One of the basic tenets of evolution is mutation driven by natural selection and COMPETITION. There is no competition when working on pure strains of organisms in tightly controlled conditions. Competition would be provided by a heterogenous mix of species competing for an energy source, such as in the digestive system.So if life first appeared on Earth 3 or 4 billion years ago, why isn't there a gradual increase in complexity such as what darwinian evolution would predict?
Instead, we have a massive leap in evolution during a period of time that is relatively short compared to the amount of time life has existed on Earth.
One human generation = about one million bacterial generations, yet despite Scientists trying their best to provoke evolution in such creatures using methods like irradiation and who knows what else over the course of numerous decades, they still haven't succeeded in making bacteria anything other than what they already were.
Still the same creationist tripe. It still doesn't address the main issues that everyone has brought up already; that flagellar proteins need not have been evolved solely for flagellar purposes; and that not all of them are necessary for function, since even crude motility is better than no motility for a bacterium.
Note that the Cambrian explosion does not necessitate the complete evolution of all that life. All it requires is the evolution and spread of something that makes these organisms apparent in the fossil record.
Your painstaking differentiation is strange. Because not even Judge Jones, given the best evidence that "intelligent design" proponents could give in a court of law, could tell the difference between creationism and intelligent design. If you really aren't Christian, you're fooling yourself. Plain and simple.
Irreducible complexity is an asinine argument with which to try to refute evolution with. Just because a system is completely dependent on the sum of its parts, doesn't mean it couldn't have been achieve through iteration. Let me give you a simple example: picture a jenga tower that is perfectly built. Every block except those that are necessary for support have been removed and the tower is "perfectly" tall. This tower now can no longer support the elimination of any further bricks, that would cause failure. That system is irreducibly complex; however, it doesn't mean it wasn't able to be assembled, or created from less complex parts.While I have no formal training/education in genetics, I can still form an opinion, and I respect that you seem to know more about this subject than I do.
However, your explanation about the genes used to make flagella mirrors the supposed rebukes of Behe's ID that I've read, in that it side steps or glosses over the fundamental difficulties involved in explaining how evolution could create such parts via numerous, slight, successive changes over long periods of time.
This is a very recent article in which the author attempts to show why Behe hasn't been refuted on the flagella.
It's rather technical in nature, but perhaps you could fully understand it.
I've found that things are not always as simple as they appear, and that darwinian evolutions have a tendency to trivialize things. You offer a simple and generalized explanation, but I can't help but think why it wouldn't occur to Behe (and many others) who has a PhD in Molecular Biology that it really is as simple as it appears to be.
Don't know and don't care too much either. I wrote that for you, as you claimed to have little background.I wonder how Jonathan M, the man who wrote that article would respond to your assertions.?
It's not my specialty, but I've got a pretty good background in bacterial transcriptional regulation. You're wrong about how it "had" to evolve. There's nothing about sigma factors that is outlawed by a darwinian theory. Despite what the article is telling you, sigma factors aren't strictly necessary for transcription in bacteria. Really. They're pretty close to it now, but not completely. They help and it's pretty easy to see how RNA polymerase became (mostly) dependent on them - it's pretty much a specific example of my previous post.Any way, the reason why your explanation doesn't make sense to me, is that it doesn't account for the fact that for mutation to be responsible for such an elaborate and well ordered system (if you read the article, synethesis of the flagellum is dependent on a "tightly ordered cascade in which expression of one gene at a given level requires the prior expression of another gene at a higher level") as bacterial flagella, all the right mutations (and none of the harmful ones) would have to occur at the same time without mistakes, instead of the numerous, slight successive changes ascribed to by darwinian evolution.
"Well connected?" WTF does that even mean?Natural selection would not select parts/components that weren't already well connected and regulated now would it?
Especially when according to the article, "untimely expression of flagellum proteins may induce a strong immune response in the host system, something no bacterium wants to do."
No.So for darwinian evolution to create bacterial flagella the way you explain it, would require it to not be subject to chance or random probability.
Strawman. Again, there's no reason for the apparent record to show a linear or smooth increase in complexity. There's no reason from theory to assume that the actual increase in complexity should be linear or smooth.
You're demanding that labs growing around 10e7 or so bacteria per generation, in under 10e2 years, should do something similar to what was seen in I dunno, 10e21 organisms, in hugely diverse niches, at a time span well over 10e7? You're really trying to make a direct comparison between these two things?
Why wouldn't it? Nirvana fallacy.You're seemingly contradicting yourself here. Why would natural selection choose parts that weren't already well ordered, and completely functional?
Bacteria have no immune system. They have enzymes that protect against viral infection, but that is no immune system. And it does not do anything to actual proteins.Crude motility just wouldn't cut I think, nor half measures, and as the article I linked to states, "untimely expression of flagellum proteins may induce a strong immune response in the host system, something no bacterium wants to do."
Argument from personal incredulity.So there is an evolutionary reason behind the tight and well ordered regulation of bacterial flagella.. If you read about the nature of the flagellum and how complex and well regulated their synthesis is, it's inconceivable that an unintelligent process such as random mutation could be responsible for their making.
Where on earth did you get the idea that random mutations needed to be slight? And where did you get the idea that the phenotypic changes produced by even slight modifications needed to be slight?First off, random mutation relies on slight, successive modifications OVER TIME, as well as probability in combination with natural selection. How could a random process create such an elaborate system, where in even the slightest mistakes results in a complete and utter failure?
Half-function is better than no function. That is all that is relevant. Nirvana fallacy. Again. It's like saying that because cars are less efficient than teleportation we should all abandon our vehicles and just walk everywhere.Natural selection wouldn't choose half functional proteins that didn't work properly, and endangered the existence of the organism? It wouldn't allow the genes responsible for such to pass a long to future generations..
Why not?But why did all these organisms appear suddenly during the Cambrian period? What about all the billions of years before the Cambrian period, when Life was already present on Earth?
c.f. Kitzmiller et al. vs. Dover Area School District.The difference between ID and creationism is quite plain and easy to understand.. Also, you have no idea who I am or what my beliefs are, yet you're telling me I'm fooling myself by not calling myself a Christian?
In some aspects...yes. In other aspects...nowhere near close. And yes, you're fooling yourself. Because in terms of detail, creationism is just another creation myth. Pure and simple. You don't have to be Christian to believe in a creation myth. But tagging yourself onto a Christian ideology because you don't have the education necessarily to critically appraise the literature on a scientific theory...that is dumb.Thats a remarkable statement. I'm the World's foremost expert on myself, and I'm telling you that I'm not a Christian.
I have no idea since Jesus is likely a figment of man's imagination. ()![]()
Umm... because, it's not intelligent. It's spontaneous, mutation driven.You're seemingly contradicting yourself here. Why would natural selection choose parts that weren't already well ordered, and completely functional?
You did read the word may, right?Crude motility just wouldn't cut I think, nor half measures, and as the article I linked to states, "untimely expression of flagellum proteins MAY induce a strong immune response in the host system, something no bacterium wants to do."
Inconcievable to you and that "scientist" who likes to use big mol bio words, maybe. Apparently not to many people in this thread or in the scientific community.So there is an evolutionary reason behind the tight and well ordered regulation of bacterial flagella.. If you read about the nature of the flagellum and how complex and well regulated their synthesis is, it's inconceivable that an unintelligent process such as random mutation could be responsible for their making.
Yes it does, all the damn time. You're telling me that genetic diseases don't get passed from parent to child? Look up cystic fibrosis.Natural selection wouldn't choose half functional proteins that didn't work properly, and endangered the existence of the organism? It wouldn't allow the genes responsible for such to pass a long to future generations..
Yes it does, all the damn time. You're telling me that genetic diseases don't get passed from parent to child? Look up cystic fibrosis.
Yes, thank you for the refresher of bio 101. CF and sickle cell anemia are shown benefits of the heterozygote advantage; however, my point in the context of refuting his arguement is that let's say tomorrow malaria was eradicated somehow, 1000 years from now, the sickle cell anemia allele would continue to pass on and its carriers would continue to express less than functional - or "half functional" to use his wording - hemoglobin. According to his faulty understanding of natural selection this wouldn't be possible, which is wrong.His point was that if a genetic disease caused nothing but disease, it would disappear rapidly, because of natural selection. This is correct.
So, why then, are some genetic disorders so common - e.g. cystic fibrosis, sickle cell disease and many others? For example, in Europe, about 6% of people carry the cystic fibrosis gene.
The answer is because most of these genetic disorders come from total loss of the gene - higher organisms, have a 2nd copy of the gene (1 from each parent). If one if faulty, in some cases, the other copy is sufficient for health.
If the loss of one copy caused no effect, you would still expect the faulty gene to disappear, but very slowly. But, what if having one copy of the 'faulty' gene was advantageous? This would provide natural selective pressure to keep the gene in the population. So, do these genes like cystic fibrosis and sickle cell confer an advantage, if present together a normal backup copy?
The answer is yes. Diarrhea is one of the most common causes of death in the world. The cystic fibrosis gene, when working correctly, is used for pumping fluid from the blood into body cavities (like the intestines) and elsewhere (sweat). People with one faulty copy are therefore expected to get less severe diarrhea, particularly if they get a severe infection, like cholera (where cholera toxin blocks the normal control system that regulate fluid pumping into the intestine, causing the body's fluid pump systems to run maxed out and causing severe dehydration). Similarly, the sickle cell gene is a faulty form of hemoglobin. With 2 dud copies, the blood cells are severely defective. However, with 1 dud, they aren't too bad - except, when they get infected with malaria parasites, where the small concentration of dud hemoglobin is toxic to the parasites, and the presence of the gene is therefore strongly favored by natural selection - even if someone unfortunate to inherit 2 defective copies of the gene is extremely unfit.
Aren't you worried about going to hell for saying something like that? I'm pretty sure that's against your rules.I have no idea since Jesus is likely a figment of man's imagination. ()![]()
...and the point of Ophir's (and my) argument is that even half-functional proteins confer some advantage to an organism compared to no protein at all.His point was that if a genetic disease caused nothing but disease, it would disappear rapidly, because of natural selection. This is correct.
So, why then, are some genetic disorders so common - e.g. cystic fibrosis, sickle cell disease and many others? For example, in Europe, about 6% of people carry the cystic fibrosis gene.
The answer is because most of these genetic disorders come from total loss of the gene - higher organisms, have a 2nd copy of the gene (1 from each parent). If one if faulty, in some cases, the other copy is sufficient for health.
If the loss of one copy caused no effect, you would still expect the faulty gene to disappear, but very slowly. But, what if having one copy of the 'faulty' gene was advantageous? This would provide natural selective pressure to keep the gene in the population. So, do these genes like cystic fibrosis and sickle cell confer an advantage, if present together a normal backup copy?
The answer is yes. Diarrhea is one of the most common causes of death in the world. The cystic fibrosis gene, when working correctly, is used for pumping fluid from the blood into body cavities (like the intestines) and elsewhere (sweat). People with one faulty copy are therefore expected to get less severe diarrhea, particularly if they get a severe infection, like cholera (where cholera toxin blocks the normal control system that regulate fluid pumping into the intestine, causing the body's fluid pump systems to run maxed out and causing severe dehydration). Similarly, the sickle cell gene is a faulty form of hemoglobin. With 2 dud copies, the blood cells are severely defective. However, with 1 dud, they aren't too bad - except, when they get infected with malaria parasites, where the small concentration of dud hemoglobin is toxic to the parasites, and the presence of the gene is therefore strongly favored by natural selection - even if someone unfortunate to inherit 2 defective copies of the gene is extremely unfit.
Oh man 10,001 posts, you became a lifer in this thread! Does that mean you were born again? lol
...and the point of Ophir's (and my) argument is that even half-functional proteins confer some advantage to an organism compared to no protein at all.
Reading this thread is like listening to old cranky grannies at the old age home arguing about why that last bingo game was not fair.
The point is, the abruptness of the appearance. The cambrian explosion took place approximately 530 million of years ago. According to neo darwinian evolutionists, life first appeared on the planet approximately 3 to 4 billion years ago.
So there is an inordinate amount of time for life to evolve during that time, however the fossil records indicate that complex life forms akin to what we see today first appeared during the cambrian explosion.
So if darwinian evolution relies on slight, successive, numerous modifications occurring over lengthy periods of time, why is there such a massive gap in the fossil records?
I mean, we're talking about billions of years here..
I have been waiting for this one.The rate doesn't have to be even I agree, but there should be far more transitional stages in the fossil record before the cambrian explosion.
We're talking billions of years here, yet the fossil record doesn't indicate any sort of major or minor transitional changes that occurred during the time before the cambrian era.
