Cheesehead, the ***** removed from your grandpa's arteries was not just cholesterol, it was plague formed from modified cholesterol.
Two things can happen to cholesterol that will lead it to form arterial plaque. There's a longer explanation that involves white blood cells, but basically it amounts to this:
1) Cholesterol can become bad by being oxidized. This is perhaps why anti-oxidants can be helpful. Excess iron (a very, very powerful oxidizer -- think rust) could be a problem. Since men don't bleed every month, most men have too much iron and most men's vitamins will have no iron. However, processed foods often have extra iron, which is definitely a problem for almost all men (especially if you eat red meat) and women who are post-menopausal (they aren't bleeding either). I guess you could just give blood regularly, work with power tools a lot, join a rugby team, or maybe try leeches as well.
2) Cholesterol can become bad by being glycosylated. This is when glucose bonds to the cholesterol - a problem that is exaggerated by type II diabetes and its related diseases (hypo- and then hyper-glycemia).
Low-density lipoproteins (LDL cholesterol) is more vulnerable to oxidation or glycosylation.
In any case, eating cholesterol does not necessarily raise cholesterol. It's just not that simple. Even in cases where serum (blood) cholesterol is highly elevated, over 80% of the cholesterol is produced by the body, not the diet. High cholesterol arises when the body cannot adequately process and manage the cholesterol - overeating, pre-diabetes, overloads on difficult-to-process saturated fat, etc.. By the way, about 1/2 of coconut oil is lauric acid, a 12-carbon fatty acid. Most dietary fats are 16-18 carbons and much more difficult to break. Lauric acid is sort of pre-digested, in a sense, and is generally very easy to break down. It often does not even require extra oxygen, which most fats do.
Eggs are also not a problem. Every test with eggs that I am aware of has shown no negative impact on serum cholesterol. For healthy young men, even eating 24-eggs a day (vs. a cholesterol free egg substitute in the control), no negative impact on cholesterol was noted. Low fat diets, on the other hand, are notorious for depressing HDL (high density lipoproteins) -- the good cholesterol and raising triglyceride levels at the same time. This is a fact that Dean Ornish is quite good at hiding from his reports, even though it is clear in his data.
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