- May 11, 2008
- 19,552
- 1,194
- 126
Hi all. It has been a while. I have been rather occupied.
I hope this will help you all.
There is something i would like to share. A few years ago i wanted to start on fungi and the effects on health but i never really got the chance. Also, i was a bit to early, because only in 2018 they discovered what was needed to prove that a healthy diet is the key to a healthy life.
The SARS-CoV virus and the SARS-CoV2 virus enter human cells through the ACE-2 receptor. People who are overweight and associated with certain diseases seem to have more ACE-2 receptors. Older people in poor health may also have more ACE-2 receptors. Both groups also often are more susceptible for candida infections and then comorbidity arises. People in general with a weaker immune system can also contract various fungal infections and there may also be a case of comorbidity.
It is worth confirming or excluding all of this.
There needs to be real research because those greedy people really want to make more money than necessary by making use of the situation. The Hippocratic oath does not apply for medicine manufacturers and researchers it seems.
I'm reading about a study of the parasitic fungus Candida glabrata and that it also has ACE-2 genes. And that fungus, along with other candida family infections, is responsible for a lot of deaths every year in America and probably all over the world. It is now known that people who are extremely overweight, in the sense that it affects their health, are more likely to suffer from fungal infections. Just like that older people with a weaker immune system are more likely to suffer from fungal infections.
.
Anyway, back to the research:
When they modified this fungus so that the ACE-2 gene of the fungus no longer worked, the result was that the fungus could spread rapidly through the laboratory animal. It's very interesting and maybe microbiologists can do something with this to determine whether this has an influence on how much damage the virus SARS-CoV2 can do in people and why one person is unaffected and the other person dies.
Comorbidity is a determining factor. What if that turns out to be a candida family variant fungus?
Fungi and yeasts are much more complex than viruses. They may not mutate as quickly as viruses do. Perhaps molds and yeasts are a more interesting target than a virus.
I would also like to point out that excessive amounts of (candida) mold and yeasts in the intestines often go hand in hand with being overweight and having an extreme sugar craving and wanting to eat nonstop. Yeasts and fungi such as Candida are crazy about sugars and then the suspicion is also strongly about sucrose and high fructose corn syrup. The introduction of this type for split cheap super sugars is in the same timeline as the emergence of people with too much overweight and continuous hunger. That is again a convenient coincidence.
Some studies have been done on this in America. The yeasts and molds in the gut are known to produce neurotransmitters that can modulate the host's mental state in sufficient numbers through the bloodstream and brain. This is certainly possible with a wrong diet with extremely high carbohydrates and hardly any exercise. In other words, they are more parasitic fungi and do not form a symbiotic alliance with the host as bacteria do in the gut flora and fauna, body orifices and on the skin. This is from research that has only been known for 3 years that fungi and yeasts live in the gut and are suppressed by symbiotic bacteria.
For more information, see the links :
The research from 2004.
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence
Het mycobioom en de invloed op onze eigen darm-hersenas
Niet alleen bacteriën van het microbioom, ook schimmels en gisten beïnvloeden onze darm-hersenas.
www-bonusan-com.translate.goog
"
The Mycobiome: A Neglected Component in the Microbiota-Gut-Brain Axis
"
The Mycobiome: A Neglected Component in the Microbiota-Gut-Brain Axis
"
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence
"
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence
I hope this will help you all.
There is something i would like to share. A few years ago i wanted to start on fungi and the effects on health but i never really got the chance. Also, i was a bit to early, because only in 2018 they discovered what was needed to prove that a healthy diet is the key to a healthy life.
The SARS-CoV virus and the SARS-CoV2 virus enter human cells through the ACE-2 receptor. People who are overweight and associated with certain diseases seem to have more ACE-2 receptors. Older people in poor health may also have more ACE-2 receptors. Both groups also often are more susceptible for candida infections and then comorbidity arises. People in general with a weaker immune system can also contract various fungal infections and there may also be a case of comorbidity.
It is worth confirming or excluding all of this.
There needs to be real research because those greedy people really want to make more money than necessary by making use of the situation. The Hippocratic oath does not apply for medicine manufacturers and researchers it seems.
I'm reading about a study of the parasitic fungus Candida glabrata and that it also has ACE-2 genes. And that fungus, along with other candida family infections, is responsible for a lot of deaths every year in America and probably all over the world. It is now known that people who are extremely overweight, in the sense that it affects their health, are more likely to suffer from fungal infections. Just like that older people with a weaker immune system are more likely to suffer from fungal infections.
.
Anyway, back to the research:
When they modified this fungus so that the ACE-2 gene of the fungus no longer worked, the result was that the fungus could spread rapidly through the laboratory animal. It's very interesting and maybe microbiologists can do something with this to determine whether this has an influence on how much damage the virus SARS-CoV2 can do in people and why one person is unaffected and the other person dies.
Comorbidity is a determining factor. What if that turns out to be a candida family variant fungus?
Fungi and yeasts are much more complex than viruses. They may not mutate as quickly as viruses do. Perhaps molds and yeasts are a more interesting target than a virus.
I would also like to point out that excessive amounts of (candida) mold and yeasts in the intestines often go hand in hand with being overweight and having an extreme sugar craving and wanting to eat nonstop. Yeasts and fungi such as Candida are crazy about sugars and then the suspicion is also strongly about sucrose and high fructose corn syrup. The introduction of this type for split cheap super sugars is in the same timeline as the emergence of people with too much overweight and continuous hunger. That is again a convenient coincidence.
Some studies have been done on this in America. The yeasts and molds in the gut are known to produce neurotransmitters that can modulate the host's mental state in sufficient numbers through the bloodstream and brain. This is certainly possible with a wrong diet with extremely high carbohydrates and hardly any exercise. In other words, they are more parasitic fungi and do not form a symbiotic alliance with the host as bacteria do in the gut flora and fauna, body orifices and on the skin. This is from research that has only been known for 3 years that fungi and yeasts live in the gut and are suppressed by symbiotic bacteria.
For more information, see the links :
The research from 2004.
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence
Het mycobioom en de invloed op onze eigen darm-hersenas
Niet alleen bacteriën van het microbioom, ook schimmels en gisten beïnvloeden onze darm-hersenas.
"
The Mycobiome: A Neglected Component in the Microbiota-Gut-Brain Axis
"
The Mycobiome: A Neglected Component in the Microbiota-Gut-Brain Axis
"
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence
"
Inactivation of Transcription Factor Gene ACE2 in the Fungal Pathogen Candida glabrata Results in Hypervirulence