- Jul 16, 2001
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The authors say their findings "support the hypothesis that the induction of angiogenesis and VEGF expression by ethanol represent an important mechanism of cancer progression associated with alcoholic beverage consumption."
Recent data in a cellular model has demonstrated that ethanol increases cellular production of VEGF, an important signaling protein in blood vessel growth, particularly in tumors. Jian-Wei Gu. M.D. from the University of Mississippi Medical Center and colleagues further investigated the possible mechanism between ethanol-induced blood vessel growth and VEGF using a chick embryo model. The investigators exposed chick embryos inoculated with fibrosarcoma cells to saline or physiologically relevant levels of ethanol for nine days.
The authors say their findings "support the hypothesis that the induction of angiogenesis and VEGF expression by ethanol represent an important mechanism of cancer progression associated with alcoholic beverage consumption."
Recent data in a cellular model has demonstrated that ethanol increases cellular production of VEGF, an important signaling protein in blood vessel growth, particularly in tumors. Jian-Wei Gu. M.D. from the University of Mississippi Medical Center and colleagues further investigated the possible mechanism between ethanol-induced blood vessel growth and VEGF using a chick embryo model. The investigators exposed chick embryos inoculated with fibrosarcoma cells to saline or physiologically relevant levels of ethanol for nine days.
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